Elemental Green Tea is designed for maximum boost to energy levels as well as a potent fat burner. Green Tea stimulates thermogenesis in brown adipose tissue and reduces absorption of dietary fats.
Product Details:
It prolongs the actions of norepinephrine by preventing its breakdown and it also acts as an appetite suppressant. Green Tea also possesses strong anti-oxidant properties making this product one of Elemental Nutrition’s most popular. Take 2 - 4 tablets daily, either as tablets or drop 1 tablet into a glass or cup of hotwater and drink as tea. Each Tablet Contains Green Tea Extract 10:1 700mg, Quercetin 3 Rutinose 200mg, Epigallo catechin gallate (egcg) 200mg, CONTAINS CAFFEINE.
Research:
Mechanism for green tea's anti-cancer action revealed!
5/17/2005 - Green tea appears to protect against cancer by
affecting a 'promiscuous' protein that pharmaceutical
experts are already targeting in their work on anti-cancer
drugs, according to new research.
The study, by PhD student Christine Palermo at the
University of Rochester Medical Center, reveals a potential
new mechanism to explain the tea, and particularly its
active compounds' action against cancer.
While many studies suggest that green tea protects people
against some forms of cancer, such as breast and liver
cancer, exactly how it does so has been difficult to
pinpoint.
"It's important to find out the source of green tea's
protective effects," said toxicologist Thomas A Gasiewicz,
whose work on the harmful effects of dioxin led the
Rochester group to explore the protective effects of green
tea.
"What is exciting here is that a completely new mechanism
has been found that very well could be responsible for its
protective effects, and that could help us find a compound
that is much more potent."
Palermo, Gasiewicz, and current undergraduate Claire
Westlake discovered that a chaperone protein known as HSP90 is involved in conferring green tea's protective effects.
Other researchers have shown that many cancer cells have an increase in the level of HSP90 compared to healthy cells,
and that when HSP90 is blocked, levels of proteins that make
cancer cells grow drop.
Drug makers are currently working on ways to block HSP90,
which is known as a promiscuous chaperone protein because it binds to many different cells and receptors in the body. It turns out that those researchers are trying to duplicate what green tea does naturally. Gasiewicz says green tea might modulate HSP-90 in a way that researchers have not seen before.
Gasiewicz and his group have shown how dioxin and other
substances like cigarette smoke manipulate a major cancer-
causing molecule, the aryl hydrocarbon (AH) receptor, which
frequently plays a role in turning on genes that are
oftentimes harmful.
Two years ago the team discovered that AH activity is
inhibited by a chemical found in white and green teas,
epigallocatechingallate or EGCG.
"We initially hypothesized that EGCG would work in the same
way as other AH antagonists, by binding directly to it. We
were completely surprised that this isn't the case," said
Gasiewicz, whose work was funded by the National Institute
of Environmental Health Sciences and the American Institute
for Cancer Research.
Instead, the team found that EGCG binds to HSP90, a protein
that helps other proteins stay stable, serving the same role
as a tail on a kite. When the two bind, HSP90 no longer
turns on the AH receptor, stopping the cascade of events
that would lead to the activation of several harmful genes.
Another potential mechanism for green tea's action was
outlined recently by a team at the University of Wisconsin
and CaseWestern ReserveUniversity in Cleveland, Ohio.
They found that green tea polyphenols reduce levels of
insulin-like growth factor-1 (IGF-1) in prostate tumour
cells in a mouse model for human prostate cancer. Increased
levels of IGF-1 are associated with higher risk of several
cancers, such as prostate, breast, lung and colon.
Meanwhile, researchers at KyushuUniversity in Japan
reported last year that EGCG appears to inhibit tumour cell
growth by binding to a receptor on cells called the 67-kDa
laminin receptor. A variety of tumours produce abnormally
high levels of 67 LR, and the receptor is thought to be
involved in the spread of cancers through the body.
The new research was published in "Biochemistry".
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